FBI billboards not about Assata Shakur; it’s about repressing the black community
May 5, 2013
Following the ludicrous announcement that the Obama administration has placed Assata Shakur on its “most wanted terrorist list”, the FBI has erected billboards in Newark, New Jersey announcing its recently increased $2 million dollar reward. However, any critically thinking person knows that these billboards are not about capturing Assata Shakur but sending a message to the rest of us. Interestingly, perhaps just a coincidence or not, Newark, New Jersey is the place where a theater co-owned by Shaquille O’Neil, recently reneged on an agreement to show a popular independent film about the life of another former member of the Black Panther Party for Self Defense, political prisoner Mumia Abu-Jamal.
Is Assata Shakur in New Jersey? No, she is not and the FBI and the Obama administration know exactly where she is, in Cuba where she has lived since being granted political asylum by its government in 1979 after escaping from prison.
This is not about Assata Shakur, it is about sending a message to the Black community and those that live within it who stand up to police violence, oppression and murder of residents, one of the very reasons for the formation of the Black Panthers. It is about the political repression of those who advocate on the behalf of the many political prisons being held by the United States government often in torturous conditions. It is about sending a message to anyone who would take up arms in defense of life, liberty and true freedom in a country that is home to the largest prison population in the world which the federal government and various corporations use as slave labor. It is about sending a message to those that would dare stand up and point out that the US government is the most violent entity on the planet and one that commits acts of terrorism against non-white people and nations on behalf of maintaining the American imperialist status-quo.
Why else would the U.S. government seek to name Assata Shakur as a domestic terrorist after all these decades? We are talking about a woman who was shot twice while attempting to give herself up to police who were co-operating with Federal authorities to target and assassinate or otherwise eliminate members of the Black Liberation movement just as they had done and admitted in a civil lawsuit to doing to Martin Luther King Jr.
The FBI and its corporate media wing fail to report the details of the sham case built against Assata Shakur after failing to win convictions on other trump up charges. The corporate media is failing to point out that a police officer, a state witness against Assata Shakur for the murder of another police officer, has recanted his testimony and admitted to lying on the stand. Medical personnel stated that because of nerves severed by a bullet, Assata Shakur would have been physically prevented from firing a weapon and it was also stated that her wounds indicate her hands were raised when she was shot consistent with her claim that she was giving herself up.
Just as Assata Shakur has pointed out that COINTELPRO utilized and received full cooperation from the corporate media to demonize and alienate freedom fighters from the people who supported them, corporate media today is still fulfilling that role. The concept of a free and independent press in America has always been a fraud and it remains so today.
The FBI caught a terrorist last week, but since he is not Muslim, you probably didn’t hear about it on the news.Talbot is a white, radical right-wing conservative who was arrested by the FBI on charges of “attempted interference with commerce by robbery, solicitation to commit a crime of violence and possession of an explosive material.”
His plan was to rob banks to fund his revolution, and then also blow up mosques.
Well, since the ‘blowing up mosques’ part is more or less on the CIA’s agenda, you can see why the FBI wouldn’t want to call him a terrorist…
While there is a lot of appropriate rage about Ferguson right now, the killing of John Crawford, III is getting less attention than it deserves. I put Shaun King’s tweets and history lesson on the matter in chronological order for easier consumption.
You really should be following Shaun King on Twitter.
Darren Hunt of Utah
The murder of young Black Men by police continues.
oh for fucks SAKE
Y’all he was shot in the back…. HE WAS SHOT IN THE BACK…
He was carrying a sword? This mf in my geography class carried a sword to class everyday and when I expressed my discomfort it was dismissed. But this brotha was shot in the back.
and it was a blunted sword.. couldn’t have cut anybody… but white people walking around with loaded rifles in target…
Exactly! This is evil.
Damn. Niggas can’t even cosplay anymore? I would love to see the cosplay community say something about this but that definitely won’t happen
Also: this paper is edited by a clown. It should’ve been in the first fucking paragraph that this dude was cosplaying. I’m reading this shit wondering why the fuck this negro is walking down the street with a sword and obvious answer is hidden almost at the en of the article.
This dude was cosplaying.
He was dressed up in a costume.
Should all black people just stay home on Halloween this year?
Friendly reminder that the police shot a black cosplayer in the back
New research shows that schizophrenia isn’t a single disease but a group of eight genetically distinct disorders, each with its own set of symptoms. The finding could be a first step toward improved diagnosis and treatment for the debilitating psychiatric illness.
The research at Washington University School of Medicine in St. Louis is reported online Sept. 15 in The American Journal of Psychiatry.
About 80 percent of the risk for schizophrenia is known to be inherited, but scientists have struggled to identify specific genes for the condition. Now, in a novel approach analyzing genetic influences on more than 4,000 people with schizophrenia, the research team has identified distinct gene clusters that contribute to eight different classes of schizophrenia.
“Genes don’t operate by themselves,” said C. Robert Cloninger, MD, PhD, one of the study’s senior investigators. “They function in concert much like an orchestra, and to understand how they’re working, you have to know not just who the members of the orchestra are but how they interact.”
Cloninger, the Wallace Renard Professor of Psychiatry and Genetics, and his colleagues matched precise DNA variations in people with and without schizophrenia to symptoms in individual patients. In all, the researchers analyzed nearly 700,000 sites within the genome where a single unit of DNA is changed, often referred to as a single nucleotide polymorphism (SNP). They looked at SNPs in 4,200 people with schizophrenia and 3,800 healthy controls, learning how individual genetic variations interacted with each other to produce the illness.
In some patients with hallucinations or delusions, for example, the researchers matched distinct genetic features to patients’ symptoms, demonstrating that specific genetic variations interacted to create a 95 percent certainty of schizophrenia. In another group, they found that disorganized speech and behavior were specifically associated with a set of DNA variations that carried a 100 percent risk of schizophrenia.
“What we’ve done here, after a decade of frustration in the field of psychiatric genetics, is identify the way genes interact with each other, how the ‘orchestra’ is either harmonious and leads to health, or disorganized in ways that lead to distinct classes of schizophrenia,” Cloninger said.
Although individual genes have only weak and inconsistent associations with schizophrenia, groups of interacting gene clusters create an extremely high and consistent risk of illness, on the order of 70 to 100 percent. That makes it almost impossible for people with those genetic variations to avoid the condition. In all, the researchers identified 42 clusters of genetic variations that dramatically increased the risk of schizophrenia.
“In the past, scientists had been looking for associations between individual genes and schizophrenia,” explained Dragan Svrakic, PhD, MD, a co-investigator and a professor of psychiatry at Washington University. “When one study would identify an association, no one else could replicate it. What was missing was the idea that these genes don’t act independently. They work in concert to disrupt the brain’s structure and function, and that results in the illness.”
Svrakic said it was only when the research team was able to organize the genetic variations and the patients’ symptoms into groups that they could see that particular clusters of DNA variations acted together to cause specific types of symptoms.
Then they divided patients according to the type and severity of their symptoms, such as different types of hallucinations or delusions, and other symptoms, such as lack of initiative, problems organizing thoughts or a lack of connection between emotions and thoughts. The results indicated that those symptom profiles describe eight qualitatively distinct disorders based on underlying genetic conditions.
The investigators also replicated their findings in two additional DNA databases of people with schizophrenia, an indicator that identifying the gene variations that are working together is a valid avenue to explore for improving diagnosis and treatment.
By identifying groups of genetic variations and matching them to symptoms in individual patients, it soon may be possible to target treatments to specific pathways that cause problems, according to co-investigator Igor Zwir, PhD, research associate in psychiatry at Washington University and associate professor in the Department of Computer Science and Artificial Intelligence at the University of Granada, Spain.
And Cloninger added it may be possible to use the same approach to better understand how genes work together to cause other common but complex disorders.
“People have been looking at genes to get a better handle on heart disease, hypertension and diabetes, and it’s been a real disappointment,” he said. “Most of the variability in the severity of disease has not been explained, but we were able to find that different sets of genetic variations were leading to distinct clinical syndromes. So I think this really could change the way people approach understanding the causes of complex diseases.”
»american psycho« by mimi cabell and jason huff
this book was made by sending the entire text of bret easton ellis’ american psycho between two gmail accounts page by page. we saved the relational ads for each page and added them back into the text as footnotes. in total, we collected over 800 relevant ads for the book. the constellations of footnoted ads throughout these pages retell the story of american psycho in absence of the original text. this retelling reveals gmail’s unpredictable insensitivity to violence, racism, and sex. it serves as a blurry portrait of an algorithm that exists in our everyday communication simultaneously forming a new portrait of the lead character, patrick bateman.
jason huff/mimi cabell nyc 2012
Apple download - https://itunes.apple.com/us/app/aspire-news/id733163167?mt=8
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I’m sorry - not my usual stuff - but this is incredibly important. Please share around. It could save someone’s life.
I DON’T CARE IF THIS DOESN’T SUIT YOUR BLOG TYPE IF YOU DON’T REBLOG THIS I’M JUDGING YOU